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FSGS: Focal segmental glomerulosclerosis

Notably, mitochondrial dysfunction induces endoplasmic reticulum (ER) stress, excessive reactive oxygen species (ROS) production, aberrant autophagy, apoptosis, and disrupted calcium ion homeostasis, culminating in inflammatory imbalance via activated signaling cascades [13,14,15,16,17]

Early surgical intervention when local recurrence is detected enhances survival, because the dismal outcomes are associated with distant metastasis

(1993) observed that only a complete mixture of Ceramides, Cholesterol, and free fatty acids in an equimolar ratio (1:1:1) facilitated normal barrier recovery