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Furthermore, given the fact that danger signals often change multiple times in lipid metabolism, developing more potent activators/inhibitors that target multiple FAO signaling pathways, including the CPT system or combined with other anti-inflammatory/antitumor therapeutics, may facilitate translation of this promising strategy into clinical application

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Part I: technological concepts and evolution

USPs 20 and 33, but not their inactive forms, increase the cAMP signals obtained upon repeated stimulation of cells after an initial agonist treatment, suggesting that USPs 20 and 33 also promote efficient resensitization of 2 ARs [87]