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Studies suggest the peptide may induce lipolysis without significantly affecting insulin or IGF-1 levels in laboratory models, which would, in principle, reduce the glucose-intolerance risk associated with full HGH exposure

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By blocking NNMT activity, the compound preserves nicotinamide availability for the NAD+ salvage pathway, leading to 1.2- to 1.6-fold increases in intracellular NAD+ concentrations

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