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A separate set of enzymes and transporters also plays a buffering role by eliminating acetyl-CoA from inside mitochondria created by the pyruvate dehydrogenase complex that is in excess of its utilization by the Krebs cycle

Acylcarnitine profile Metabolites that are abnormal in VLCAD deficiency patients are usually C12, C14, C14:1, C14:2 and C16 [43], with C14:1 accumulation as most important marker, which is derived from partial breakdown of oleic acid (C18:1w9)

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It suggests that R254X in Lebanese patients shared the same ancestor as Chinese patients

Wine, beer, alcohol and polyphenols on cardiovascular disease and cancer

3A, One-way ANOVA followed by Tukeys post hoc test), ruling out the possibility of a direct inhibitory activity on catalase