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Notoginsenoside R1-loaded mesoporous silica nanoparticles targeting the site of injury through inflammatory cells improves heart repair after myocardial infarction

(2) overproduction of GDH in INS-1E cells and in rat and mouse pancreatic islets increased GSIS at high glucose concentrations, suggesting GDH is a rate-limiting step in GSIS [69]

AtkinsonAHillSLBarangeMPakhomovEARaubenheimerDSchmidtKet al

Biomedicines , 10 (11), 2696

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This rapid neutralization prevents ROS from tearing electrons away from healthy cellular structures, effectively putting out the biochemical fires driving chronic inflammation