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Glucuronidation of macelignan by human liver microsomes and expressed UGT enzymes: Identification of UGT1A1 and 2B7 as the main contributing enzymes

Abraham Arimuko, MD

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Animal experiments Four-week-old male BALB/C nude mice were purchased from the animal center of Changsheng Biotechnology (Liaoning, China), and kept in a specific pathogen-free condition and randomly divided into groups

Moreover, leptin treatment in obese subjects has proven to be ineffective

396 Early-onset and juvenile-onset POLG -related disorders generally result from biallelic pathogenic variants with autosomal recessive inheritance, whereas late-onset disorders (mainly PEO) may arise from a heterozygous POLG pathogenic variant with autosomal dominant inheritance