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There are a range of perturbations that result in the loss of proteostasis, including genetic mutations that alter amino acid sequences, errors during protein synthesis and post-translational modifications (PTMs), dysfunction of the molecular chaperones responsible for assisting protein folding, and various cellular stresses, such as heat shock, oxidative stress, and nutrient deprivation

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Nevertheless, regardless of the certain background of primary HT, the common feature of pathological findings in the thyroid gland is its lymphocytic infiltration