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Biochemical compounds that were higher in the associated diets are more likely to be contributing to this disease, but a smaller number of compounds that were lower also could contribute

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Jan 2018;24(1):37-47

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Its effect indicates how PPAR and C/EBP could be downregulated for GHG to inhibit preadipocyte maturation into mature adipocytes, hence controlling excessive body fat production [36]

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