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Therefore, in addition to antioxidant properties, and increasing GSH levels, NAC protects against A toxicity through activation of anti-apoptotic signaling pathways

Collectively, these studies demonstrate the potential of MSNs as multifunctional platforms for therapeutic and theragnostic pursuits

doi: 10.1128/mBio.00470-17

4.3.4 Organelle dysfunction CS also impairs the function of critical organelles central to ferroptosis regulation: (1) Mitochondrial Damage: CS stimulates DRP1-dependent mitochondrial fission, resulting in fragmented, dysfunctional mitochondria that overproduce reactive oxygen species (ROS)

This approach holds the potential to precisely and effectively eradicate tumors while minimizing damage to healthy and non-cancerous cells

[DOI] [PMC free article] [PubMed] [Google Scholar] Cabral M