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Causal effect of neurodegenerative diseases on antioxidant biomarkers We performed reverse MR analysis to evaluate the potential causal effects of neurodegenerative diseases on biomarkers associated with antioxidants

The five most upregulated genes were aldol-keto reductases (maximum 30.2 fold), alcohol dehydrogenases (maximum 11.65 fold) and nitrate reductase (8.06)

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Recombinant human IGF-I was first available for experimental therapy in the late 1980s, what allowed the development of long-term studies in children with severe primary IGF-I deficiency (defined as a height SDS and IGF-I SDS less than or equal to 3 and normal or elevated GH levels)[375]

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Protonation of the oxidant accelerates the reaction because the protonated form is a better electrophile, which is an opposite effect to the aforementioned favorable deprotonation of the thiol