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V., Douat C., Rosu F., Mergny J.-L., Salgado G

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The p.R778L mutation affects transmembrane transport of copper while p.G943S and p.M769V mutations result in defective copper metabolism but preserved ceruloplasmin levels

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Modern studies show why: it neutralizes free radicals directly, flips on the Nrf2 switch, and spares cysteine, the key raw material for glutathione

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X-ray crystal structure of a recombinant human myoglobin mutant at 2.8 A resolution

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[DOI] [PubMed] [Google Scholar] 102.Lescure A., Rederstorff M., Krol A., Guicheney P., Allamand V

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