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They show that glutathione binds to a surface cavity, promoting assembly of the viral capsid, and providing a potential therapeutic target.

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These effects were further amplified when GSH-depleted cells were treated with ASA

To determine whether increased GSH levels would modulate EEG synchronization, N-acetyl-cysteine (NAC), a glutathione precursor, was administered to patients in a randomized, double-blind, crossover protocol for 60 days, followed by placebo for another 60 days (or vice versa)

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