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The aim of this review is to collate what is currently known about LCACs as effectors of cardiac function and to provide a pathophysiological basis for the recent metabolomic interest of circulating LCACs in the plasma of CVD patients

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Thus, blocking iron-induced lipid oxidation is important for MUFA-induced longevity (although salinazid could also extend lifespan via other iron-dependent processes or its effect on lipid droplet number)

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