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However, neither molecular mechanisms responsible for the APE/Ref-1 gene expression induction upon oxidative injury nor functional data regarding the real in vivo role played by post-translational modifications in controlling APE/Ref-1 functional activities are clear.24 Understanding the possible relationships existing between APE/Ref-1 levels and oxidative-stress-based human liver pathologies is of enormous importance, not only for the comprehension of the role and mechanism that APE/Ref-1 may play in the initiation and development of these pathologies but also for developing diagnostic markers and therapeutic tools

albicans will also initiate carbon metabolism remodeling independent of SEF1

Mao S, Xiang W, Huang S, Zhang A

Evaluation and optimization of sample pretreatment for GC/MS-based metabolomics in embryonic zebrafish

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