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Though the mechanism for DMF function is still not fully understood, published reports support the therapeutic potential of DMF in oxidative stress regulation in alleviating SCD symptoms
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Individuals with the rs662 AA genotype have a lower lung cancer risk in non-smokers and lower levels of the oxidative stress marker 8-OHdG, suggesting that PON1 gene polymorphisms co-regulate lung cancer occurrence and oxidative stress levels in conjunction with environmental factors [252]
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