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6E), suggesting that L-cystine reduction contributes to cell death upon combined STF-31 and BSO treatment

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In an -naphthylisothiocyanate-induced mouse model with cholestatic liver injury, CGA suppresses cell death and neutrophilic and monocytic infiltration and reverses dysregulated hepatocyte transporters and enzymes related to synthesis, uptake, metabolism, and efflux of bile acids [43,174]

Emerging interventions aim to restore immune function through metabolic and inflammatory regulation