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Microglia-specific overexpression of -synuclein leads to severe dopaminergic neurodegeneration by phagocytic exhaustion and oxidative toxicity

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Gut bacterial therapies that improve insulin sensitivity by altering the composition of the gut microbiota have become a new therapeutic modality

The most accepted hypothesis centres around the failure of self-tolerance which occurs inside the thymus in AChR-MG

Since the natural GLP-1 is degraded after a few minutes, pharmaceutical companies have produced medicines that are more long-lasting and replicate the original effect

However, understanding how these medications interact and complement each other is essential before starting dual therapy