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And it appears endogenous GLP-1 does just that, it helps locally and temporarily to help suppress T-cell inflammation, the problem is that the half-life of endogenous GLP-1 is just a few minutes, so any systemic effects are somewhat muted

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What's the continuity-of-care plan if compounded GLP-1 availability changes due to FDA regulatory action

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provide clear evidence that GLP-1 signaling in the CNS is also linked to the control of peripheral glucose homeostasis by inhibiting noninsulin-mediated glucose uptake by muscle and increasing insulin secretion from the pancreas