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Conclusions GLP-1 agonists reduce fetal survival, weight, growth and skeletal ossification and can induce major congenital anomalies

All patients gave a written informed consent and the human sample collection was approved by the Ethics Committee of Zhongshan Hospital, Fudan University

For researchers, this regulatory uncertainty creates practical questions

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Key GLP1 therapies are integrated into national insurance schemes, and the countrys aging population underscores its strategic importance in diabetes and obesity management

Monoacylglycerol binds to GPR119 in L cells to promote the release of GLP-1, GIP and insulin ( 2+ were observed following GPBA-R activation in single GLUTag cell, and bile acid and GPBA-RA increased GLP-1 secretion in intestinal cultures (Parker et al., 2012b)