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It does so by stimulating insulin release and reducing glucagon, among other actions

This gives your body the time it needs to recover and adapt, helping you maintain muscle, boost energy, and achieve sustainable progress

Additionally, obesity and IR both contribute to -cell dysfunction and apoptosis, mechanistically explained by glucotoxicity and increased immunogenicity (the accelerator hypothesis) or by -cell exhaustion due to increased metabolic demand (the overload hypothesis) [23]

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