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Therefore, we speculated that T cells could induce microglial polarization through direct contact, cytokine-mediated communication or antigen presentation, thereby amplifying their impact on the cerebral immune milieu

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In contrast, compoundspecific effects are distinct for each DPP4i, influenced by their unique chemical compositions and pharmacokinetic profiles

The increased BSH activity was probably due to the regulatory action of A

GLP-1s require ongoing possibly lifelong injections to maintain results
