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Supplementary material The Supplementary Material for this article can be found online at: References 1

Therefore, it is conceivable that IIH represents a distinct neurological manifestation of this adverse metabolic phenotype with female androgen excess and male androgen deficiency conferring a risk factor for the development of IIH
In an embryofetal development study in pregnant cynomolgus monkeys, subcutaneous doses of 0.015, 0.075 and 0.15 mg/kg twice weekly (1.9-, 9.9- and 29-fold the MRHD) were administered throughout organogenesis, from Gestation Day 16 to 50
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