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Matza LS, Boye KS, Stewart KD, et al

What evidence is needed to prove a GLP-1 drug injury

By reducing the expression of pro-inflammatory cytokines and activating anti-inflammatory signaling, GLP-1RA-based therapies may mitigate the inflammatory processes that sustain neuropathic pain [66,67,68]

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Such effects could plausibly involve relaxation of blood vessels, improved renal salt handling and reduced stress signalling, all of which may contribute to lower blood pressure

Cardiac pericyte reprogramming by MEK inhibition promotes arteriologenesis and angiogenesis of the ischemic heart