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We treat GLP 1 medications as one tool in a comprehensive metabolic and hormonal plan not a standalone solution

PI3K activates protein kinase B (Akt), which inactivates several important substrate proteins, such as glycogen synthase kinase 3 (GSK-3)18 and forkhead box O,19 and activates mammalian target of rapamycin (mTOR).20 As a result, it modulates some cellular processes, such as cell survival, proliferation, apoptosis, protein synthesis, inflammation, ER stress, mitochondrial function, and autophagy in neurodegenerative disorders.1820 Akt also promotes B-cell lymphoma 2 and B-cell lymphoma extra-large transcription by activating cyclic adenosine monophosphate (cAMP) response element-binding protein (CREB).21 Thereafter, it regulates learning, memory,22 and neuron survival.23 MAPK regulates various cellular activities including proliferation, differentiation, apoptosis or survival, inflammation, and innate immunity.24 Impairment of insulin signaling is common in diabetes-related NDs