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Regarding chronic exposure, cases of cardiac fibrosis [91], increased cardiac inflammation [92], and enlarged left ventricle [84] have been identified in preclinical models exposed to e-cigarette aerosol for three to eight months

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Silencing of NLRP3 also abrogated the release of LDH and the increase of PI positive cells, indicating an inhibition of the cell lysis and pyroptotic cell death by nicotine (Fig

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