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Nisin, an apoptotic bacteriocin, induces ChaC1 activation, calcium influx and cell cycle arrest in the G2 phase, leading to increased apoptosis and decreased cell proliferation in vitro and in vivo (68)

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Dimeric PFKM interacts with nucleosomes and phosphorylates histone H3 at serine 10 (H3S10), functioning as a protein kinase to promote mitosis and cell proliferation

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In extracellular Ca 2+ -depleted medium (Figure 2E) or in the presence of 100 M lanthanum ions (La 3+ ), a non-selective calcium channel blocker, tBuOOH-induced [Ca 2+ ] i accumulation was completely inhibited (not shown), suggesting the occurrence of Ca 2+ entries under oxidative conditions