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doi: 10.1080/19490976.2021.1994833 200 ZhuL
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Structural analysis of the wild-type HsaGST A2-2 and HsaGST A3-3 shows that the steroid substrate is not binding to HsaGST A2-2 in an orientation compatible with ketosteroid isomerase activity, apparently explaining the different activities of the two enzymes (Tars et al., 2010)
however, both the age of onset and disease penetrance vary considerably among different mutations and across endemic regions [6, 7]
Ara, arabinose
