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I.PodyachevaE.RudenkoA.TsygankovA

451 In NCOR1 KO mice, nicotinamide adenine dinucleotide phosphate (NADPH) consumption for de novo fatty acid synthesis can occur, and redox homeostasis can be maintained by activating the G6PD-dependent pentose phosphate pathway (PPP)

Given that stimulation of T cells changes the balance of ferroptosis-promoting and protecting events, we subsequently employed inhibitors of GPX4, as a primary regulator protecting cells from ferroptosis, to determine T cells sensitivity to ferroptosis

Nishimura, M

[DOI] [PubMed] [Google Scholar] 61.Rius-Prez S., Torres-Cuevas I., Milln I., Ortega .L., Prez S