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[DOI] [PMC free article] [PubMed] [Google Scholar] 257.He J, Zhou D, Yan B

217 In short, based on the fact that cell proliferation-related receptors are overexpressed in tumor cells but are not expressed or underexpressed in normal cells, NDCDS can be designed to target the corresponding receptors and then actively target tumors to release drugs

doi: 10.1021/acsnano.4c18636
This effect was reversed in sB-treated keratinocytes (Fig

273,274,275 P300/CREB-binding protein (CBP) is one such protein

Brucein D, a naturally occurring quassinoid, enhances the sensitivity of pancreatic ductal adenocarcinoma (PDAC) cells to gemcitabine (GEM) by promoting the ubiquitin-proteasome-dependent degradation of NRF2 and suppression of the NRF2 signaling pathway [104]
