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Mitochondrial Mg2+ homeostasis decides cellular energy metabolism and vulnerability to stress

Using ImageStream analysis, we observed live cells harbouring GFP-expressing S

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BSO treatment causes a significant reduction of the total GSH in liver (70 %), which is attributable to the diminished levels of the reduced GSH (GSH, 71 %) in rats [12]

Glutathione acts as a primary shield, neutralizing these molecules before they can cause significant damage

Following cerebral ischemia, EA treatment activates the Wnt/-catenin pathway enhancing proliferation of neural progenitor cells [117]