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In addition, the expression of TRPV 1, for example, is proved in trigeminal neurons and brain regions engaged in migraine, and what is more, using its agonists provides increased calcium influx and, as a result, CGRP and substance P release, which initiates the cascade of neuroinflammation in migraine [62]

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TMAO can upregulate scavenger receptors, such as cluster of differentiation 36 (CD36) and scavenger receptor class A type 1 (SR-A1), and promote the formation of foam cells, leading to development and progression of atherosclerosis (63)

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