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FIGURE 3 Further, emerging evidence has indicated that copper stabilizes the HIF-1 protein through inhibiting prolyl hydroxylases-mediated prolyl hydroxylation in an iron-independent manner, which is required for transcriptional activation of HIF-1 of a series of target genes (Martin et al., 2005

[DOI] [PubMed] [Google Scholar] 219.Madiha S, Haider S

[DOI] [PubMed] [Google Scholar] 65.Van Epps H.L

Therefore, cancer cells may be more susceptible to ALT activation due to the binding of epitalon to the already reduced levels of histone H1, whereas normal cells may be more resilient as they naturally express higher levels of H1

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