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In an experimental model, transgenic mice expressing a kinase-deficient IGF-1R -subunit (thus displaying reduced signal transduction in both IGF-1R and hybrid receptors) developed diabetes early on life [162, 236]

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When these mice where administered IGF-1 they showed lowered fasting glucose, and because no functional IGF-1R is present in muscle, such effect is believed to be due to renal gluconeogenesis suppression [129, 163]

Research community reports describe first measurable body composition changes at weeks 4 to 6, with peak effect at weeks 8 to 12

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Available from: fact sheet_diet_en.pdf