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CONCLUSION DISCLOSURE OF INTERESTS REFERENCES Ayer A, Tan SX, Grant CM, Meyer AJ, Dawes IW, Perrone GG

The staining specificity was tested by omission of primary antibodies

[20] This repression supports the self-renewal and proliferation of neural stem cells

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EGFR, a key tyrosine kinase receptor, regulates critical growth factors involved in tumor proliferation and survival

This modulation is achieved through its regulation of inflammatory signaling pathways and its ability to enhance the expression of M2 macrophage markers, such as CD206 and arginase-1, while simultaneously suppressing M1-associated cytokines, including TNF- and IL-6 ( 3.2.3 Modulation of T Cell subset balance in immune responses by quercetin Currently, there is no direct evidence in the literature indicating that quercetin can treat AP by modulating the balance of T cell subsets ( 3.3 Advantages and challenges of quercetin in clinical applications 3.3.1 Comparison of quercetin with conventional therapies for acute pancreatitis Conventional therapies for AP, such as N-acetylcysteine (NAC) and corticosteroids, primarily target specific disease components but fail to offer a comprehensive solution for the multifactorial pathogenesis of the disease ( In contrast, quercetin, a flavonoid with potent antioxidant and anti-inflammatory properties, offers a broader therapeutic approach by simultaneously addressing oxidative stress and inflammation