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Whether the accumulation of a specific type of lipid is sufficient and necessary for causing insulin resistance is unclear

[DOI] [PMC free article] [PubMed] [Google Scholar] 156.Gade P, Kimball AS, DiNardo AC, et al

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rather, it can support a more personalized conversation with your provider about whether lipotropic therapy fits your metabolic profile and whether compounded GLP-1 or metabolic support options merit exploration

Notably, Laurent et al.(51) recently demonstrated that liver-targeted recombinant AAV8 (rAAV8) gene therapy effectively rescued LAL-D in a Lipa/ mouse model, correcting lipid accumulation and normalizing gene expression patterns and mitochondrial function, suggesting rAAV8-mediated liver gene transfer may be an effective curative approach