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Conclusion In this review, we have provided an extensive overview of the main questions that remain unanswered regarding the molecular pathogenesis of SCA3, and what does the extensive and high-quality research so far teach us on the more likely initial events that compromise cellular function

Crucially, this diabetes-induced gut microbiota dysbiosis may profoundly adversely affect the ENS and impede GI motility, as evidenced by recent mechanistic investigations ( Figure 2 )

Doing so can increase the risk of serious side effectsfor example, fat-soluble vitamins (such as vitamin A, vitamin D, vitamin E, and vitamin K) can accumulate in the body over time, reaching toxic levels

10.1016/j.pharmthera.2007.07.006 7 Alescio-LautierB.PabanV.Soumireu-MouratB

However, clinical data in humans remains limited