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Further in support of this include the emergence of sexbased differences in pain sensitivity around puberty [129], fluctuations in pain responses across phases of the menstrual cycle in women [130, 131], and the higher prevalence of adverse childhood experiences among females, a factor that may independently contribute to increased risk [132]

Some research suggests Epithalon might modulate epigenetic regulators that control TERT expression, including DNA methylation patterns and histone modifications

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