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& Y.Sa) and a Grant-in-Aid for Scientific Research(C) (to Y.Sa.)

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In IDH1 mutant glioblastoma, BCAT1 is transcriptionally suppressed because of the hypermethylation of three CpGs in the promoter, and this is also the consequence of IDH1 mutation-induced 2-hydroxyglutarate (2-HG) (Tonjes et al., 2013)

These results clearly indicate that the observed induction of c-Ret phosphorylation by hHGF and aML5-PEG3 relies on Met phosphorylation, although we are not certain which factor(s) present in FBS plays a role in this transactivation event