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In models of Achilles tendon transection, the peptide accelerated healing and restored biomechanical properties to near-normal levels
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fmr1 mutant PyrN at middle exhibited a moderate reduction in dendrite arbor size, whereas fmr1 mutant PyrN at right exhibited severely impaired dendrite growth
Perimenopause and underactive thyroid symptoms often overlap
Liposomal formulations show modest improvements but still fall far below IV-achievable levels
