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In such severe cases, cerebral oedema becomes clinically apparent 12 hours to 4 days after the overdose [18,20,50,87], although CNS depression may be delayed if a slow-release preparation has been ingested [12,50]

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These bioactive metabolites, such as short-chain fatty acids (SCFAs), trimethylamine (TMA), trimethylamine N-oxide (TMAO), bile acids (BAs), and lipopolysaccharides (LPS), are implicated in conditions such as hypertension and atherosclerosis

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