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Posttranslational modification may alter the conformation of the p53 antioncogene which suppresses its antiproliferative function in acute myelogenous leukemia and in growth factor stimulated normal hematopoietic cells

Sagara et al

PMID: 36516757 Front Immunol CD38+CD39+ NK cells associate with HIV disease progression and negatively regulate T cell proliferation

In the AMY 1 R and AMY 3 R Y37 rAmy interacts with residues in the respective RAMP and the CTR ECD, however the small side chain of P37 Cagri is completely buried by the surrounding CTR ECD residues, forming extensive hydrophobic contacts with W79 ECD and no direct interactions with either RAMP1 (AMY 1 R) or RAMP3 (AMY 3 R), again consistent with the restrictive interactions formed by Pro32 sCT with the CTR ECD

Pro-apoptotic protein BAD: Expression and phosphorylation in leukemia

Similarly, in another study, Seta et al