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Toxigenic Clostridium difficile PCR ribotypes from wastewater treatment plants in Southern Switzerland

Morscher et al

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Iron overload reduces glucose oxidation in murine cardiac muscle, and this is accompanied by decreased activity of mitochondrial complexes I-IV and low ATP production [36]

Initially characterized as a mitochondrially localized pro-apoptotic factor, in vitro and in vivo experiment reveal that myristoylation-mediated translocation to the plasma membrane converts FSP1 into a potent ferroptosis inhibitor ( 2 ), which directly quenches lipid radicals through its antioxidant activity ( FSP1 expression is directly transcriptionally regulated by Nrf2, while Nrf2s downstream target NAD(P)H quinone dehydrogenase 1 (NQO1) cooperatively maintains the reduced state of CoQ10 ( 3.4 Mitochondria in ferroptosis Mitochondria, central to eukaryotic cellular iron metabolism, maintain iron homeostasis primarily through heme and ferritin storage

Specifically, the proportion of Car4-positive cells increased from 1.07% at ambient conditions (23) to 3.72.% under cold exposure in young mice, while this increase was even more obvious in aged mice, with Car4-positive cells rising from 2.20% at 23 to 6.42% under cold exposure (Fig