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Pathogenesis of autoimmune disease

During the 1990s and early 2000s, research interest increased in peptide fragments capable of interacting with cellular signalling systems and molecular regulatory pathways

The accumulation of iron in the brain may induce lipid peroxidation through oxidative stress, potentially triggering ferroptosis

PLGA segments prevented the initial burst release of NO as protecting layer and it had the important role for NO-releasing behavior of NPs

Retinoids inhibit measles virus through a type I IFN-dependent bystander effect

Acknowledgements We thank all members of the Birsoy laboratory for helpful suggestions