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While it did at first appear surprising that p53 but not ATM was required for reducing histone levels in response to chronic irradiation, we noted that p53 protein was upregulated, with concomitant increase of its downstream target p21, in chronically irradiated RPE-1 cells independently of ATM status (Fig
doi: 10.1016/j.neuroscience.2021.03.009
Assay of radical scavenging activity of antidotes against chemical warfare agents by DPPH test using sequential injection technique
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Choosing between glycolysis and oxidative phosphorylation: a tumors dilemma
