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2018 Sep 25;8(3):121-124

Campos Pacheco J

Mitochondria-targeted drugs can activate the cGAS-STING pathway, promote DAMPs release, induce ferroptosis, and regulate immune cell activity by producing MTROS, reducing m, triggering endoplasmic reticulum stress or Lipid peroxidation, etc

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Antibodies Following primary antibodies were used in this study: rabbit polyclonal anti-BDNF (1:1000, #ab226843, Abcam), rabbit monoclonal anti-TrkB (1:1000, #4603, Cell Signaling Technology), anti-p75 NTR (1:1000, #4201, Cell Signaling Technology), anti-phospho-TrkA (Tyr674/675)/TrkB (Tyr706/707) (1:1000, #4621, Cell Signaling Technology), anti-p44/42 MAPK (Erk1/2) (1:1000, #4695, Cell Signaling Technology), anti-phospho-p44/42 MAPK (Erk1/2) (Thr202/Tyr204) (1:1000, #4377, Cell Signaling Technology), anti-Akt (pan) (1:1000, #4691, Cell Signaling Technology), anti-phospho-Akt (Ser473) (1:1000, #4058 Cell Signaling Technology), anti-BAD (1:1000, #9239, Cell Signaling Technology), anti-phospho-BAD (Ser136) (1:1000, #4366, Cell Signaling Technology), anti-phospho-GSK-3 (Ser9) (1:1000, #9323, Cell Signaling Technology), rabbit polyclonal anti-phospho-TrkB (Tyr816) (1:1000, #NBP1-03499SS, Novus Biologicals), anti-phospho-TrkB (Tyr515) (1:1000, # PA5-36695, Thermo Fisher Scientific), anti-Caspase-9 Antibody (1:1000, #9502, Cell Signaling Technology), mouse monoclonal anti--Tubulin (1:1000, #2125, Cell Signaling Technology)