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The drug is an N-protected dipeptide and can be written as Pyz-Phe-boroLeu, which stands for pyrazinoic acid, phenylalanine, and leucine with a boronic acid instead of a carboxylic acid

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Because peptides may influence tissue repair and Tissue Regeneration, improper use could theoretically promote unwanted cellular activity rather than regenerative wellness

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Table 1 Animal studies evaluating the anti-inflammatory effect of GLP-1 in atherosclerosis

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Clinically used small molecule drugs of ROCK signaling Netarsudil (N) and Wnt signaling LY2090314 (L) were also tested

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