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The molecular mechanisms underlying this neuronal loss are linked to mitochondrial dysfunctions that increase ROS levels, which can interfere with the enzymatic processes of oxidative deamination of dopamine

For example, during the luteal phasewhen estrogen levels peaksome studies have observed higher glutathione concentrations in the body

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The characterization of the specific binding of [ 3 H]- N -acetylaspartylglutamate to rat brain membranes

The following potential mechanism is proposed: l-carnitine prolongs the ability of endothelial cells to regulate blood flow during ischaemia, and an increase in serum carnitine concentration enhances capillary endothelial function [38]