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While GAD67 is a hydrophilic cytoplasmic protein, GAD65 undergoes post-translational hydrophobic modifications, which enable it to be anchored on the membrane compartments of the synaptic vesicles (Kanaani et al., 1999, 2002)

These findings strengthen the hypothesis of a causal role for increased homocysteine levels in cardiovascular disease

That can be unsettling, especially when the medication is supposed to help you function better, not make it harder to get through the day

10.1016/J.NEUINT.2024.105760 Neurochem

However, whether these in vivo effects are directly caused by MGO or MGO associated BBB integrity loss and damage in other cell types is unsure

Beyond editing, strategies are being developed to selectively remove large portions of mutated mtDNA by reconstituting double-strand break repair pathways within mitochondria, potentially eliminating deletion-bearing genomes (171)