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Description

This alteration suppressed GATA3 expression via the bile acid-Takeda G protein-coupled receptor 5(TGR5)-cyclic adenosine monophosphate (cAMP) signaling pathway, leading to a marked ~90% reduction in IL-22 secretion from intestinal group 3 innate lymphoid cells (ILC3s), which subsequently induced insulin resistance, ovarian dysfunction (characterized by increased follicular cysts and decreased corpora lutea), and impaired fertility (82)

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Several enzymatic and non-enzymatic defence mechanisms that guard cells against free radical damage have been identified in different cellular localizations, including mitochondria, plasma membrane, endoplasmic reticulum, peroxisomes, and cytosol

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Further studies have found that IPF ECM inhibited miR-29 expression upstream at the transcriptional level, and suppressed Dicer1 downstream at the processing steps to maintain the fibrosis progression ( Cells and Regulators Epithelial Cells Damage Currently, the prominent initiation of IPF is widely considered to be repeated lung epithelial cell damage and repair dysfunction