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Lastly, gene expression analysis demonstrated that the molecular fingerprint of pathological cardiac hypertrophy was partly reversed by -MSH with a significant reduction in TAC-induced expression of fibrosis-related genes including Col1a2 (collagen type I, alpha 2) and Mmp2 (matrix metalloproteinase-2) (Appendix Fig
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& Gold, M.H
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DOI.org (Crossref) , VCV000003597.87 ClinVar NCBI
